Corrigendum: Molecular pathogenesis of Chlamydia trachomatis (Frontiers in Cellular and Infection Microbiology, (2023), 13, (1281823), 10.3389/fcimb.2023.1281823)

Abstract

In the published article, there were errors in some sections of the text where we referred to the wrong gene, citation or did not clearly articulate previous findings. These are detailed below: A correction has been made to Section 2 Transcription and regulatory factors as a target to understand pathogenesis, paragraph 3 where we said phase-locked σ54 mutants were used instead of CtcC mutants to investigate the transcriptional profiles for the σ54 regulon. This sentence previously stated: The correct sentence appears below: A correction has been made to Section 3.3 Inc proteins, paragraph 1 and 3 where we erroneously referred to IncS instead of CTL0390 and we have provided further clarity on previous findings on Inc proteins. This section previously stated: “Inc proteins, or inclusion membrane proteins, are characterised by the presence of SNARE-like motifs in their coding sequence and have a diversity of functions. “…”IncA is required for inclusion vacuole fusion (Table 1B), other functions of Inc proteins include; host cell viability (IncG), establishing infection (IncD) and promoting replication (IncV).” …”Here, we focus on recent IncM, IncS and CpoS which have been recently characterised.” The corrected section appears below: “Inc proteins or inclusion membrane proteins, share bilobed hydrophobic domains enabling anchoring to the inclusion membrane. Some Incs contain the presence of vesicle-targeting SNARE-like motifs in their coding sequence (e.g. IncA), which may facilitate host-pathogen interactions (Cingolani et al., 2019; Paumet et al., 2009).”…”IncA is required for inclusion vacuole fusion (Table 1B), other functions of Inc proteins include; a possible role in maintaining host cell viability (IncG) (Scidmore and Hackstadt, 2001; Verbeke et al., 2006) and as an early Inc, IncD may be important for establishing the inclusion as a replicative niche (Shaw et al., 2000), although further studies are required to confirm this.”…”Here, we focus on IncM, CTL0390 and CpoS which have been recently characterised.” A correction has been made to Section 3.4 ChlaDUB, paragraph 1 where the sentence erroneously stated that ChlaDUB1 has been shown to stabilize GLUT-1 and GLUT-3, but the referenced study only showed ChlaDUB1 stabilizes GLUT-1. This sentence previously stated: The corrected sentence appears below: A correction has also been made to Section 6 Plasmid, paragraph 1 where the sentence erroneously stated pGP3 as a transcriptional regulator instead of pGP4. This sentence previously stated: The corrected sentence appears below: The authors apologize for these errors and state that this does not change the scientific conclusions of the article in any way. The original article has been updated.